Nature 2003, 423:309–312.PubMedCrossRef 37. Antony E, Tomko EJ, Xiao Q, Krejci L, Lohman TM, Ellenberger T: Srs2 disassembles Rad51 filaments by a protein-protein interaction triggering ATP turnover

and dissociation of Rad51 from DNA. Mol Cell 2009,35(1):105–115.PubMedCrossRef 38. Sung P: Catalysis of ATP-dependent homologous DNA pairing and strand exchange by yeast RAD51 protein. Science 1994,265(5176):1241–1243.PubMedCrossRef 39. Bai Y, Davis AP, Symington LS: A novel allele of RAD52 that causes severe DNA repair and recombination deficiencies only in the absence RGFP966 ic50 of RAD51 or RAD59 . Entospletinib clinical trial Genetics 1999, 153:1117–1130.PubMed 40. Jablonovich Z, Liefshitz B, Steinlauf R, Kupiec M: Characterization of the role played by the RAD59 gene of Saccharoymces cerevisiae in ectopic recombination. Curr Genet 1999, 36:13–20.PubMedCrossRef 41. Bailis AM, Maines S, Negritto MT: The essential helicase gene RAD3 suppresses short-sequence recombination in Saccharomyces cerevisiae . Mol Cell Biol 1995,15(5):3998–4008.PubMed 42. Liefshitz B, Parket A, Maya R, Kupiec M: The role of DNA repair genes

in recombination between repeated sequences in yeast. Genetics 1995, 140:1199–1211.PubMed 43. Rong L, Klein HL: Purification https://www.selleckchem.com/products/apr-246-prima-1met.html and characterization of the SRS2 DNA helicase of the yeast Saccharomyces cerevisiae . J Biol Chem 1993,268(2):1252–1259.PubMed 44. Rong L, Palladino F, Aguilera A, Klein HL: The hyper-gene conversion hpr5–1 mutation of Saccharomyces cerervisiae is an allele of the SRS2/RADH gene. Genetics 1991, 127:75–85.PubMed 45. Palladino F, Klein HL: Analysis of mitotic and meiotic defects in Saccharomyces cerevisiae SRS2 DNA helicase mutants . Genetics 1992,132(1):23–37.PubMed 46. Morrison DP, Hastings PJ: Characterization of the mutator mutation mut5–1. Mol Gen Genet 1979,175(1):57–65.PubMedCrossRef 47. Lopes J, Ribeyre C, Nicolas A: Complex minisatellite rearrangements generated in the total or partial absence of Rad27/hFEN1 activity occur in a single generation

and are Rad51 and Rad52 dependent. Mol Cell Biol 2006,26(17):6675–6689.PubMedCrossRef 48. Freudenreich CH, Kantrow SM, Zakian VA: Expansion and length-dependent fragility of CTG repeats in yeast. Science 1998,279(853):853–856.PubMedCrossRef 49. Johnson RE, Kovvali GK, Prakash L, Prakash S: Role of yeast Rth1 selleck products nuclease and its homologs in mutation avoidance, DNA repair, and DNA replication. Curr Genet 1998, 34:21–29.PubMedCrossRef 50. Fasullo MT, Davis RW: Direction of chromosome rearrangements in Saccaromyces cerevisiae by use of his3 recombinational substrates. Mol Cell Biol 1988,8(10):4370–4380.PubMed 51. Nguyen HD, Becker J, Thu YM, Costanzo M, Koch EN, Smith S, Myers CL, Boone C, Bielinsky AK: Unligated Okazaki fragments induce PCNA ubiquitnation and a requirement for Rad59-dependent replication fork progression. PLoS One 2013,8(6):e66379.PubMedCrossRef 52.